‘백신 부작용’ 다룬 학술 논문… 알고 보니 129편이나 있었다

Fact


▲아이는 태어난 지 18개월 만에, 백신을 맞고 자폐증에 걸렸다. ▲그러나 질병예방통제센터는 “백신과 자폐증 사이에는 아무런 상관관계가 없다”고 발표했다. ▲엄마는 이 말을 믿을 수가 없었다. ▲엄마는 그날부터 ‘백신과 자폐증의 연관성’을 입증할 논문을 찾기 시작했다. ▲결과는 놀라웠다. ▲무려 129편의 논문을 찾아낸 것. ▲엄마는 인터넷에 논문 리스트를 공개했다. ▲광고없는 언론 팩트올이 해당 논문 129편의 목록을 정리했다.


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미국 메인 주에 사는 48세 여성 진저 테일러(Ginger Taylor)에게는 정상 생활이 힘든 아들 챈들러(Chandler, 15)가 있다. 챈들러는 태어난 지 18개월 때였던 2003년 백신을 맞았다. 이후 챈들러는 사람을 똑바로 쳐다보지 못하고, 말을 제대로 하지 못하게 됐다. 자폐증에 걸린 것이다. 평범한 주부였던 엄마 테일러는 그날 이후 백신 반대 운동가로 변신했다.


미국 CDC(질병예방통제센터)는 유아들에게 ‘백신 접종’을 권하고 있다. 2015년에는 “백신과 자폐증 사이에 상관관계가 없다”고 발표했다. 하지만 테일러는 “백신과 자폐증의 연관성을 뒷받침하는 연구 결과들이 많이 있다”고 반박했다. 그는 그 증거로 무려 129편의 논문을 온라인 문서공유 사이트인 ‘스크립드(Scribd)’에 올렸다. 이들 논문은 백신의 부작용과, 백신 성분이 인체에 미치는 영향에 대해 다룬 것들이다.


문서 공유사이트에 129편 논문 올려


2014년 환경독성과 약리학 학회지(Environmental Toxicology and Pharmacology)에 실린 논문은 “수은이 자폐증을 일으키는 주요 원인”이라고 지적했다. 수은은 백신의 보존제로 사용되는 유기화합물 ‘티메로살(Thimerosal)’의 구성성분이다. 미국 텍사스 대학 건강과학센터는 2008년 “수은 등 적은 양의 유독물질에 장기간 노출되면 자폐증과 같은 발달 장애에 걸릴 위험이 높아질 수 있다”고 주장했다.


수은 외의 다른 성분도 문제로 지목됐다. 독성과 환경건강학 저널(Journal of toxicology and environmental health)은 2011년 “백신에서 수은이 제거돼도 자폐증을 유발하는 다른 성분이 존재할 수 있다”는 논문을 실었다. 같은해 무기생화학 저널(Journal of Inorganic Biochemistry)에는 “백신의 알루미늄 성분과 자폐증 사이에는 인과관계가 있을 가능성이 있다”는 논문이 실렸다.


“자폐증 뿐 아니라 면역질환도 일으켜”


백신이 자폐증 뿐만 아니라 다른 질병까지 유발한다는 연구결과도 있다. 미국 국립생물정보센터는 2005년 “백신을 맞은 뒤에 일시적으로 자가면역질환이 발생한다”는 논문을 수록했다. 자가면역질환이란 갑상샘염, 류머티즘 등 면역기능의 이상으로 발생하는 병을 뜻한다.


미국의학협회저널(JAMA)은 2004년 “홍역, 볼거리, 풍진 혼합백신인 MMR백신을 접종하자 열성 경련(열과 함께 경련이 발생하는 것)의 발병률이 높아졌다”는 논문을 공개했다.


“의사들은 백신과 관련된 연구결과 잘 몰라”


테일러는 미국 지역지인 ‘포틀랜드 프레스 헤럴드(Portland press herald)’에 “나는 (백신에 대한) 자료를 모으면서, 환자에게 조언해주는 의사들 대부분이 백신에 대한 연구결과를 잘 알지 못한다고 확신하게 됐다”고 말했다.


테일러가 공개한 ‘백신 부작용 논문’ 129편의 목록은 다음과 같다.


Increased risk of developmental neurologic impairment after high exposure to thimerosal-containing vaccine in first month of life. 


Hepatitis B vaccination of male neonates and autism diagnosis


Gender-selective toxicity of thimerosal


Mercury toxicokinetics-dependency on strain and gender


Review of the differences in developmental, Psychiatric, and Medical Endophenotypes between Males and Females with Autism Spectrum Disorder 


Mercury toxicity; Genetic susceptibility and synergistic effects


Autism; a form of lead and mercury toxicity


Do aluminium vaccine adjuvants contribute to the rising prevalance of autism?


Administration of aluminium to neonatal mice in vaccinerelevant amounts is associated with adverse long term neurological outcomes.


AluminumInduced Entropy in Biological systems:Implications for Neurological disease


A comparison of temporal trends in United States autism prevalance to trends in suspected environmental factors


Toxic Metals and Essential Elements in Hair and Severity of Symptoms among Children with Autism


Assessment of infantile mineral imbalances in autism spectrum disorders(ASD)


Abnormal measles-mumps-rubella antibodies and CNS autoimmunity in children with autism.


Infection, vaccines and other environmental triggers of autoimmunity


Impact of environmental factors on the prevalence of autistic disorder after 1979


A Positive Association found between Autism Prevalence and Childhood Vaccination uptake across the U.S. Population


Neonatal administration of a vaccine preservative, thimerosal, produces lasting impairment of nociception and apparent activation of opioid system in rats


19. Transcriptomic analyses of neurotoxic effects in mouse brain after intermittent neonatal administration of thimerosal


Lasting neuropathological changes in rat brain after intermittent neonatal administration of thimerosal


Persistent behavioral impairments and alterations of brain dopamine system after early postnatal administration of thimerosal in rats.


B-lymphocytes from a population of children with autism spectrum disorder and their unaffected siblings exhibit hypersensitivity to thimerosal.


Thimerosal-Derived Ethylmercury Is a Mitochondrial Toxin in Human Astrocytes: Possible Role of Fenton Chemistry in the Oxidation and Breakage of mtDNA


Thioredoxin: a novel, independent diagnosis marker in children with autism.


Inhibition of the human thioredoxin system. A molecular mechanism of mercury toxicity.


Effects of selenite and chelating agents on mammalian thioredoxin reductase inhibited by

mercury: implications for treatment of mercury poisoning.


Serological association of measles virus and human herpesvirus-6 with brain autoantibodies in autism.


Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with autism


Altered urinary porphyrins and mercury exposure as biomarkers for autism severity in Egyptian children with autism spectrum disorder


Porphyrinuria in childhood autistic disorder: Implications for environmental toxicity


An investigation of porphyrinuria in Australian children with autism.


Porphyrinuria in Korean children with autism: correlation with oxidative stress.


Uncoupling of ATP-Mediated Calcium Signaling and Dysregulated Interleukin-6 Secretion in Dendritic Cells by Nanomolar Thimerosal


The Neurobiology of Autism


Comparison of Blood and Brain Mercury Levels in Infant Monkeys Exposed to Methylmercury or Vaccines Containing Thimerosal


The retention time of inorganic mercury in the brain — A systematic review of the evidence


Brain and tissue levels of mercury after chronic methylmercury exposure in the monkey.


Interplay of glia activation and oxidative stress formation in fluoride and aluminium exposure.


Increases in the number of reactive glia in the visual cortex of Macaca fascicularis following subclinical long-term methyl mercury exposure.


Neuroglial activation and neuroinflammation in the brain of patients with autism.


Microglial Activation and Increased Microglial Density Observed in the Dorsolateral Prefrontal Cortex in Autism


Transcriptome analysis reveals dysregulation of innate immune response genes and neuronal activity-dependent genes in autism


Nanomolar aluminum induces pro-inflammatory and pro-apoptotic gene expression in human brain cells in primary culture


Aberrant NF-KappaB Expression in Autism Spectrum Condition: A Mechanism for Neuroinflammation


A Study of Nuclear Transcription Factor-Kappa B in Childhood Autism


Autism: A Brain Disorder, or A Disorder That Affects the Brain?


Activation of methionine synthase by insulin-like growth factor-1 and dopamine: a target for neurodevelopmental toxins and thimerosal.


Validation of the Phenomenon of Autistic Regression Using Home Videotapes


Blood Levels of Mercury Are Related to Diagnosis of Autism: A Reanalysis of an Important Data Set


Empirical Data Confirm Autism Symptoms Related to Aluminum and Acetaminophen Exposure


Glutathione-Related Factors and Oxidative Stress in Autism, A Review


Developmental Regression and Mitochondrial Dysfunction in a Child With Autism


Oxidative Stress in Autism: Elevated Cerebellar 3-nitrotyrosine Levels


Large brains in autism: the challenge of pervasive abnormality.


Evidence of toxicity, oxidative stress, and neuronal insult in autism.


Oxidative stress in autism.


Thimerosal neurotoxicity is associated with glutathione depletion: protection with glutathione precursors.


Toxic Metals and Oxidative Stress Part I: Mechanisms Involved in Me-tal induced Oxidative Damage


Aluminum adjuvant linked to Gulf War illness induces motor neuron death in mice.


Enrichment of Elevated Plasma F2t-Isoprostane Levels in Individuals with Autism Who Are Stratified by Presence of Gastrointestinal Dysfunction


Reduced levels of mercury in first baby haircuts of autistic children.


A case series of children with apparent mercury toxic encephalopathies manifesting with clinical symptoms of regressive autistic disorders.


Commentary: Blaxill, Baskin, and Spitzer on Croen et al. (2002), The Changing Prevalence of Autism in California


Mitochondrial Energy-Deficient Endophenotype in Autism


Briding from cells to cognition in autism pathophysiology: Biological pathways to defective brain function and plasticity 


Heavy-Metal Toxicity with emphasis on mercury 


Evidence of Mitochondrial Dysfunction in Autism and Implications for Treatment


Proximity to point sources of environmental mercury release as a predictor of autism prevalence


Epidemiology of autism spectrum disorder in Portugal: prevalence, clinical characterization, and medical conditions


Thimerosal induces neuronal cell apoptosis by causing cytochrome c and apoptosis-inducing factor release from mitochondria 


Mitochondrial mediated thimerosal-induced apoptosis in a human neuroblastoma cell line(SK-N-SH)


Possible immunological disorders in autism: Concomitant autoimmunity and immune tolerance


Influence of pediatric vaccines on amygdala growth and opioid ligand binding in rhesus macaque infants: A pilot study.


Thimerosal exposure in infants and neurodevelopmental disorders: an assessment of computerized medical records in the Vaccine Safety Datalink.


Glutathione, oxidative stress and neurodegeneration.


Hepatitis B triple series vaccine and developmental disability in US children aged 1–9 years


Induction of metallothionein in mouse cerebellum and cerebrum with low-dose thimerosal injection.


Mercury induces inflammatory mediator release from human mast cells


Blood–brain barrier and intestinal epithelial barrier alterations in autism spectrum disorders


Influence of pediatric vaccines on amygdala growth and opioid ligand binding in rhesus macaque infants: A pilot study 


Cultured lymphocytes from autistic children and non-autistic siblings up-regulate heat shock protein RNA in response to thimerosal challenge


Sorting out the spinning of autism: heavy metals and the question of incidence 


Urinary porphyrin excretion in neurotypical and autistic children.


Mitochondrial dysfunction in autism spectrum disorders: a systematic review and meta-analysis


Sensitization effect of thimerosal is mediated in vitro via reactive oxygen species and calcium signaling


86. What's going on? The question of time trends in autism 


Vaccination and Autism


Theoretical aspects of autism: Causes—A review 


Iatrogenic exposure to mercury after hepatitis B vaccination in preterm infants


Infants born late/moderately preterm are at increased risk for a positive autism screen at 2 years of age.


Preterm birth and mortality and morbidity: a population-based quasi-experimental study.


Ancestry of pink disease (infantile acrodynia) identified as a risk factor for autism spectrum disorders.


Risk factors for autistic regression: results of an ambispective cohort study.


MMR vaccination and febrile seizures: evaluation of susceptible subgroups and long-term prognosis.


MMR vaccination and febrile seizures: evaluation of susceptible subgroups and long-term prognosis.


Adverse Events following 12 and 18 Month Vaccinations: a Population-Based, Self-Controlled Case Series Analysis


Reduced GABAergic Action in the Autistic Brain.


Administration of Thimerosal to Infant Rats Increases Overflow of Glutamate and Aspartate in the Prefrontal Cortex: Protective Role of Dehydroepiandrosterone Sulfate


Neonatal Administration of Thimerosal Causes Persistent Changes in Mu Opioid Receptors in the Rat Brain


Unanswered Questions: A Review of Compensated Cases of Vaccine-Induced Brain Injury


Integrating experimental (in vitro and in vivo) neurotoxicity studies of low-dose thimerosal relevant to vaccines.


Hepatitis B vaccine induces apoptotic death in Hepa1-6 cells.


Thimerosal Induces Apoptosis in a Neuroblastoma Model via the cJun N-Terminal Kinase Pathway


Maternal thimerosal exposure results in aberrant cerebellar oxidative stress, thyroid hormone metabolism, and motor behavior in rat pups; sex- and strain-dependent effects.


The rise in autism and the role of age at diagnosis.


Slow CCL2-dependent translocation of biopersistent particles from muscle to brain


Thimerosal and autism? A plausible hypothesis that should not be dismissed.


Autism Spectrum Disorders in Relation to Distribution of Hazardous Air Pollutants in the SF Bay Area


Environmental mercury release, special education rates, and autism disorder: an ecological study of Texas.


Autism spectrum disorder prevalence and proximity to industrial facilities releasing arsenic, lead or mercury


Inflammatory Responses to Trivalent Influenza Virus Vaccine Among Pregnant Women


Elevated maternal C-reactive protein and autism in a national birth cohort.


What is regressive autism and why does it occur? Is it the consequence of multi-systemic dysfunction affecting the elimination of heavy metals and the ability to regulate neural temperature?


Neurologic adverse events following vaccination 


Immunological and autoimmune considerations of Autism Spectrum Disorders.


Identification of Unique Gene Expression Profile in Children with Regressive Autism Spectrum Disorder (ASD) and Ileocolitis


Correlations Between Gene Expression and Mercury Levels in Blood of Boys With and Without Autism


Abnormal immune response to brain tissue antigen in the syndrome of autism.


Detection and sequencing of measles virus from peripheral mononuclear cells from

patients with inflammatory bowel disease and autism.


The spectrum of ASIA: ‘Autoimmune (Auto-inflammatory) Syndrome induced by Adjuvants’


Etiology of autism spectrum disorders: Genes, environment, or both?


Thiol-modulated mechanisms of the cytotoxicity of thimerosal and inhibition of DNA topoisomerase II alpha.


Topoisomerases facilitate transcription of long genes linked to autism


Aluminum in the central nervous system (CNS): toxicity in humans and animals, vaccine adjuvants, and autoimmunity


Transcriptomic analyses of neurotoxic effects in mouse brain after intermittent neonatal administration of thimerosal.


Self-Organized Criticality Theory of Autoimmunity


Can Awareness of Medical Pathophysiology in Autism Lead to Primary CareAutism Prevention Strategies?



What is regressive autism and why does it occur? Is it the consequence of multi-systemic dysfunction affecting the elimination of heavy metals and the ability to regulate neural temperature?


Autistic Disturbance of Affective Contact



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